Prompt Description

Our approach involves a drug that, within BMSCs, binds to protein A, stabilizing its expression. This affects B RNA stability through alternative splicing. B also acts as a transcription factor, negatively regulating C, a key protective factor against ferroptosis, thereby influencing ferroptosis levels. Simultaneously, because ferroptosis affects lactate levels, it influences lactylation of D, which, through nuclear translocation and m6A modification, regulates E and F. E and F are related to osteogenesis and adipogenesis, ultimately treating age-related delayed jawbone remodeling.